July 2005
Evaluating stress response by organ and symptoms
by Dr. Howard Loomis
My last two columns
have discussed stress as a specific diagnostic entity. Clinicians should
never lose sight of the fact that the body has a very specific physiological
reaction to stress. Mechanical, nutritional, chemical, and emotional stress
all set off the same biochemical/physiological chain of events. Because this
is so, we should be able to determine whether patients are living under
excessive or continual stress and evaluate their ability to respond to
stressful events in their lives. Failure to meet the nutritional
requirements needed for specific tissues to respond to stress will result in
the appearance of symptoms.
Whenever an organ or
tissue is stressed, the amount of energy it is producing must increase.
Obviously, its nutritional needs must also increase to meet the demand. This
also increases the amount of waste that is produced proportionately and must
be removed by the extra cellular fluid.
The chart featured here
evaluates the stress response by organ and symptoms. The left‑hand column
shows symptoms that may appear when the stress has continued too long and
the organ is struggling to meet the demand. Symptoms of sympathetic
exhaustion are shown in the right‑hand column. These occur when the
organ/tissue cannot respond appropriately and nutritional exhaustion and
waste accumulation are becoming obvious.
Stress response of
the sympathetic nervous system
Stimulation of the
sympathetic nervous system increases the capability of the body to perform
vigorous muscle activity as follows:
1. Increases rates of
cellular metabolism throughout the body.
2. Shifts cellular
energy production to fat instead of glucose.
3. Increases blood
glucose concentration which can be used by the brain to provide increased
mental activity.
4. Decreases blood flow
to organs that are not needed for rapid activity.
5. Increases arterial
pressure and increases blood flow to active muscles, thus increasing
glycolysis and increasing muscle strength.
Failure of the body to
respond appropriately to the above sequence is quite common in our patients,
yet seldom fully appreciated by chiropractic or medical technicians. Today,
so much emphasis is placed on diagnosing symptoms according to the
pharmaceutical‑medical model that millions escape our recognition of their
problem until a specific disease entity can be determined. This is
unfortunate because there are many more "walking wounded" amongst us than
are diseased.
The hypothalamus
monitors events affecting the extra cellular fluid because it receives
sensory input from all tissues. It can then orchestrate a response by
signaling the autonomic nervous system via the reticulospinal tract, and the
endocrine system via its connection with the pituitary gland.
In previous columns, it
was noted that the stress response of the sympathetic nervous system occurs
when the blood glucose levels fall below normal. Among the problems that
arise when the tissues cannot respond adequately to the demand, the
pituitary is stimulated to release ACTH stimulating the adrenal cortex to
release cortisol. A cortisol deficiency results in Addison's disease.
However, before that disease occurs the body suffers from symptoms of
inflammation, i.e., heat (fever), redness, swelling, and pain. The sediment
rate increases and the patient may be diagnosed with rheumatoid arthritis,
gout, or even asthma ‑‑ all of which are treated medically by
cortisone‑related drugs.
How many of these
patients do you see before they are "branded" by the pharmaceutical‑medical
community? How many times do you identify the specific stress that is
setting off the above set of events? How many of your patients know you can
do it and prevent the devastating consequences that may follow?
(Dr. Loomis welcomes
input on the subjects covered in this column. To make a comment or ask a
question, write to him at 6421
Enterprise Lane,
Madison,
WI
53719. Visit www.loomisenzymes.com online or
call 800‑662‑2630 for information on upcoming Loomis Institute seminars.)